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Ann Saudi Med ; Vitamin D levels and left ventricular diastolic function. Open Heart ;1:e Relation of Vitamin D and parathyroid hormone to cardiac biomarkers and to left ventricular mass from the Cardiovascular Health Study. Am J Cardiol ; Vitamin D deficiency is associated with increased left ventricular mass and diastolic dysfunction in children with chronic kidney disease. Pediatr Cardiol ; Pathophysiology of anaemia: Focus on the heart and blood vessels.

Nephrol Dial Transplant ;15 Suppl Echocardiographic assessment of cardiac dysfunction in patients of end stage renal disease on haemodialysis. J Assoc Physicians India ; Left ventricular hypertrophy in African Black patients with chronic renal failure at first evaluation.

Ethn Dis ; JAMA ; Osteoporosis in populations with calcium intake: Does phosphate toxicity explain the paradox? Indian J Clin Biochem ; Left ventricular function and calcium phosphate plasma levels in uraemic patients. J Intern Med ; Mineral metabolism, mortality, and morbidity in maintenance hemodialysis. J Am Soc Nephrol ; Fibroblast growth factor 23 and mortality among patients undergoing hemodialysis.

Parathormon, calcium, phosphorus, and left ventricular structure and function in normotensive hemodialysis patients. Ren Fail ; Clin Biochem ; Prevalence and patterns of left ventricular hypertrophy in patients with predialysis chronic renal failure. J Korean Med Sci ; People who live in sunny areas at lower latitudes typically get enough vitamin D compared to people living at higher latitudes , particularly during late fall and winter.

Few foods are naturally good sources of vitamin D. The best food sources for vitamin D are fatty fish including salmon, sardines, cod, tuna and halibut. Many foods, such as some breakfast cereals and milk, are fortified with vitamin D. Milk must contain at least IU of vitamin D per cup, according to federal regulations. The practice of fortifying milk with vitamin D began in the s to prevent rickets, a bone disease that was common in children at the time. Other dairy products are not required to be fortified.

Milk substitutes such as soy milk, rice milk and nondairy creamer may or may not have added vitamin D. Too much vitamin D can be toxic. The recommended maximum intake is 25 mcg 1, IU for infants and 50 mcg 2, IU for children and adults with normal kidney function.

Your doctor can tell you if a supplement is a good choice for you and if you need it. Always check with your physician before starting an over-the-counter vitamin, mineral, diet supplement or medicine. Healthy kidneys are rich with vitamin D receptors and play a major role in turning vitamin D into its active form. This helps balance calcium and phosphorus in your body by controlling absorption of these minerals from the food you eat and regulates parathyroid hormone PTH.

Vitamin D (25(OH)D) in patients with chronic kidney disease stages

When kidneys fail, their ability to activate vitamin D is lost. Shibata, K. Association between circulating fibroblast growth factor 23, alpha-Klotho, and the left ventricular ejection fraction and left ventricular mass in cardiology inpatients.


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Tanaka, S. Association between FGF23, alpha-Klotho, and cardiac abnormalities among patients with various chronic kidney disease stages. Verkaik, M.

High fibroblast growth factor 23 concentrations in experimental renal failure impair calcium handling in cardiomyocytes. Wald, R. Correlates of left ventricular mass in chronic hemodialysis recipients. Nassiri, A. Association of serum intact fibroblast growth factor 23 with left ventricular mass and different echocardiographic findings in patients on hemodialysis. Transl Int.

Liu, E. Endocrinology , — Pastor-Arroyo, E. The elevation of circulating fibroblast growth factor 23 without kidney disease does not increase cardiovascular disease risk. FGF23 effects on the heart-levels, time, source, and context matter. Marsell, R. Gene expression analysis of kidneys from transgenic mice expressing fibroblast growth factor Xie, J. Soluble klotho protects against uremic cardiomyopathy independently of fibroblast growth factor 23 and phosphate.

Leifheit-Nestler, M. Slavic, S. Genetic ablation of Fgf23 or klotho does not modulate experimental heart hypertrophy induced by pressure overload. Experimental myocardial infarction upregulates circulating fibroblast growth factor Matsui, I. Cardiac hypertrophy elevates serum levels of fibroblast growth factor Andersen, I.

Elevation of circulating but not myocardial FGF23 in human acute decompensated heart failure. Fibroblast growth factor 23 levels are elevated and associated with severe acute kidney injury and death following cardiac surgery. Hum, J. The metabolic bone disease associated with the Hyp mutation is independent of osteoblastic HIF1alpha expression. Bone Rep. Flamme, I. FGF23 expression in rodents is directly induced via erythropoietin after inhibition of hypoxia inducible factor proline hydroxylase. Udell, J.

Fibroblast growth factor, cardiovascular prognosis, and benefit of angiotensin-converting enzyme inhibition in stable ischemic heart disease. Vascular calcification in chronic kidney disease: different bricks in the wall? Scialla, J. Fibroblast growth factor 23 is not associated with and does not induce arterial calcification.

Fibroblast growth factor and cardiovascular events in CKD. Nasrallah, M. Fibroblast growth factor FGF is independently correlated to aortic calcification in haemodialysis patients. Hunt for the culprit of cardiovascular injury in kidney disease. Arterial klotho expression and FGF23 effects on vascular calcification and function. Relationship between circulating FGF23 and total body atherosclerosis in the community. Circulating fibroblast growth factor is associated with vascular dysfunction in the community. Haring, R. Plasma fibroblast growth factor clinical correlates and association with cardiovascular disease and mortality in the framingham heart study.

Heart Assoc. Yilmaz, M.

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FGF and vascular dysfunction in patients with stage 3 and 4 chronic kidney disease. Tripepi, G. Competitive interaction between fibroblast growth factor 23 and asymmetric dimethylarginine in patients with CKD. Six, I. Direct, acute effects of Klotho and FGF23 on vascular smooth muscle and endothelium.

Phosphate and Vitamin D in Chronic Kidney Disease

Klotho modulates FGFmediated NO synthesis and oxidative stress in human coronary artery endothelial cells. Pflugers Arch. Silswal, N. FGF23 directly impairs endothelium-dependent vasorelaxation by increasing superoxide levels and reducing nitric oxide bioavailability.